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New-Onset Heart Failure

Educational Objective
Based on this clinical scenario and the accompanying image, understand how to arrive at a correct diagnosis.
1 Credit CME

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Article Information

Corresponding Author: Kenneth Tandjung, MD, PhD, Department of Cardiology, Dijklander Ziekenhuis, Waterlandlaan 250, 1441 RN Purmerend, the Netherlands (ktandjung@wlz.nl).

Published Online: March 13, 2019. doi:10.1001/jamacardio.2019.0187

Conflict of Interest Disclosures: None reported.

References
1.
Dabar  G, Harmouche  C, Habr  B, Riachi  M, Jaber  B.  Shoshin beriberi in critically-ill patients: case series.  Nutr J. 2015;14:51. doi:10.1186/s12937-015-0039-7PubMedGoogle ScholarCrossref
2.
Attas  M, Hanley  HG, Stultz  D, Jones  MR, McAllister  RG.  Fulminant beriberi heart disease with lactic acidosis: presentation of a case with evaluation of left ventricular function and review of pathophysiologic mechanisms.  Circulation. 1978;58(3, pt 1):566-572. doi:10.1161/01.CIR.58.3.566PubMedGoogle ScholarCrossref
3.
Pereira  VG, Masuda  Z, Katz  A, Tronchini  V  Jr.  Shoshin beriberi: report of two successfully treated patients with hemodynamic documentation.  Am J Cardiol. 1984;53(10):1467. doi:10.1016/S0002-9149(84)91365-1PubMedGoogle ScholarCrossref
4.
Imamura  T, Kinugawa  K.  Shoshin beriberi with low cardiac output and hemodynamic deterioration treated dramatically by thiamine administration.  Int Heart J. 2015;56(5):568-570. doi:10.1536/ihj.15-033PubMedGoogle ScholarCrossref
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