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Ventricular Arrest With a Duration of 23.8 Seconds

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1 Credit CME

A patient in their late 40s was admitted to the hospital to receive a uterine myomectomy procedure. The patient had no history of structural heart disease, hypertension, myocarditis, or sleep apnea syndrome, and denied a family history of cardiovascular disease and sudden death. The patient had not received any pharmacologic agents that would affect cardiac rhythm. Biochemical evaluation showed normal levels of whole blood cells count, electrolytes, myocardial enzymes, and brain natriuretic peptide. A 12‐lead electrocardiogram (ECG), chest radiographic imaging, and echocardiographic findings showed no abnormalities. To assess the risk of general anesthesia, the patient underwent evaluation with a Holter monitor, which recorded the patient’s cardiac activity during the syncope (Figure, A).

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A patient in their late 40s was admitted to the hospital to receive a uterine myomectomy procedure. The patient had no history of structural heart disease, hypertension, myocarditis, or sleep apnea syndrome, and denied a family history of cardiovascular disease and sudden death. The patient had not received any pharmacologic agents that would affect cardiac rhythm. Biochemical evaluation showed normal levels of whole blood cells count, electrolytes, myocardial enzymes, and brain natriuretic peptide. A 12‐lead electrocardiogram (ECG), chest radiographic imaging, and echocardiographic findings showed no abnormalities. To assess the risk of general anesthesia, the patient underwent evaluation with a Holter monitor, which recorded the patient’s cardiac activity during the syncope (Figure, A).

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Article Information

Corresponding Author: Yi Long, Chongqing Traditional Chinese Medicine Hospital, No. 7 Branch Rd, Panxi, Jiangbei District, Chongqing 400021, China (619036254@qq.com).

Published Online: July 17, 2023. doi:10.1001/jamainternmed.2023.1732

Conflict of Interest Disclosures: None reported.

References
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Lee  S , Wellens  HJ , Josephson  ME .  Paroxysmal atrioventricular block.   Heart Rhythm. 2009;6(8):1229-1234. doi:10.1016/j.hrthm.2009.04.001PubMedGoogle ScholarCrossref
2.
Zyśko  D , Gajek  J , Koźluk  E , Mazurek  W .  Electrocardiographic characteristics of atrioventricular block induced by tilt testing.   Europace. 2009;11(2):225-230. doi:10.1093/europace/eun299PubMedGoogle ScholarCrossref
3.
Sud  S , Klein  GJ , Skanes  AC , Gula  LJ , Yee  R , Krahn  AD .  Implications of mechanism of bradycardia on response to pacing in patients with unexplained syncope.   Europace. 2007;9(5):312-318. doi:10.1093/europace/eum020PubMedGoogle ScholarCrossref
4.
Hyman  MC , Papireddy  M , Frankel  DS .  Paroxysmal atrioventricular block.   JAMA Intern Med. 2021;181(8):1108-1109. doi:10.1001/jamainternmed.2021.2526PubMedGoogle ScholarCrossref
5.
Komatsu  S , Sumiyoshi  M , Miura  S ,  et al.  A proposal of clinical ECG index “vagal score” for determining the mechanism of paroxysmal atrioventricular block.   J Arrhythm. 2017;33(3):208-213. doi:10.1016/j.joa.2016.10.004PubMedGoogle ScholarCrossref
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It is the CME activity provider's responsibility to submit participant completion information to ACCME for the purpose of granting MOC credit.

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